Figure 5
From: Glucose deficit triggers tau pathology and synaptic dysfunction in a tauopathy mouse model
Glucose deprivation induces synaptic pathology in h-tau mice. (a) Representative western blot analyses of synaptophysin (SYP), post-synaptic protein-95 (PSD-95), and MAP2 in brain cortex homogenates from CTR and DG-treated h-tau mice (*P<0.05). (b) Densitometric analyses of the immunoreactivities shown in the previous panel (*P<0.05; n=9 mice per group). Results are mean±s.e.m. (c) Representative images of immunohistochemistry analyses for SYP and MAP2 in brain sections of CTR and DG-treated h-tau mice (scale bar: 100μm).
