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Akhilesh Bajpai
Sravanthi Davuluri
Kshitish Acharya
ResearchOpen Access25 Feb 2011
Nature Precedings

P: 1
A genetic-epigenetic interplay at 1q21.1 locus underlies CHD1L-mediated vulnerability to primary progressive multiple sclerosis

This study identifies distinct genetic-epigenetictranscriptional interplay at the 1q21.1 locus in relation to neuronal deficits in primary progressive multiple sclerosis (PPMS).

Majid Pahlevan Kakhki
Antonino Giordano
Maja Jagodic
ResearchOpen Access30 Jul 2024
Nature Communications

Volume: 15, P: 1-17
RADICL-seq identifies general and cell type–specific principles of genome-wide RNA-chromatin interactions

Mammalian genomes encode tens of thousands of ncRNAs that have important roles in regulation of gene expression and chromatin organization. Here, the authors present RADICLseq to map RNA-chromatin interactions in intact nuclei to shed light on these fine-tuned processes.

Alessandro Bonetti
Federico Agostini
Piero Carninci
ResearchOpen Access24 Feb 2020
Nature Communications

Volume: 11, P: 1-14
Author Correction: RADICL-seq identifies general and cell type–specific principles of genome-wide RNA-chromatin interactions

Alessandro Bonetti
Federico Agostini
Piero Carninci
Amendments and CorrectionsOpen Access19 May 2021
Nature Communications

Volume: 12, P: 1
Disease-specific oligodendrocyte lineage cells arise in multiple sclerosis

Single-cell RNA-seq of a mouse model of multiple sclerosis uncovers new oligodendrocyte populations and putative disease markers and suggests new mechanisms underlying the pathogenesis of the disease.

Ana Mendanha Falcão
David van Bruggen
Gonçalo Castelo-Branco
Research12 Nov 2018
Nature Medicine

Volume: 24, P: 1837-1844
SARS-CoV-2 promotes microglial synapse elimination in human brain organoids

Samudyata
Ana O. Oliveira
Carl M. Sellgren
ResearchOpen Access05 Oct 2022
Molecular Psychiatry

Volume: 27, P: 3939-3950

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In search of the right literature search engine(s)

A genetic-epigenetic interplay at 1q21.1 locus underlies CHD1L-mediated vulnerability to primary progressive multiple sclerosis

RADICL-seq identifies general and cell type–specific principles of genome-wide RNA-chromatin interactions

Author Correction: RADICL-seq identifies general and cell type–specific principles of genome-wide RNA-chromatin interactions

Disease-specific oligodendrocyte lineage cells arise in multiple sclerosis

SARS-CoV-2 promotes microglial synapse elimination in human brain organoids

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