Advanced search

Filter By:

Journal Check one or more journals to show results from those journals only.

Choose more journals

Article type Check one or more article types to show results from those article types only.
Subject Check one or more subjects to show results from those subjects only.
Date Choose a date option to show results from those dates only.

Custom date range

Clear all filters
Sort by:
Showing 1–5 of 5 results
Advanced filters: Author: Adam W. Whisnant Clear advanced filters

  • Here, using computational integration of multi-omics data, the authors provide a detailed transcriptome and translatome of herpes simplex virus 1 (HSV-1), including previously unidentified ORFs and N-terminal extensions. The study also provides a HSV-1 genome browser and should be a valuable resource for further research.

    • Adam W. Whisnant
    • Christopher S. Jürges
    • Lars Dölken
    ResearchOpen Access
    Nature Communications
    Volume: 11, P: 1-14

  • Herpes simplex virus 1 (HSV-1) infection disrupts transcription termination by RNA Polymerase II. Here, Djakovic et al. identify the immediate-early protein ICP22 protein of HSV-1 to induce open chromatin downstream of genes upon read-through transcription involving the histone chaperone FACT.

    • Lara Djakovic
    • Thomas Hennig
    • Lars Dölken
    ResearchOpen Access
    Nature Communications
    Volume: 14, P: 1-17

  • Herpes simplex virus-1 (HSV-1) infection disrupts transcription termination (DoTT) of host genes, but underlying mechanisms are unclear. Here, Wang et al. show that the HSV-1 immediate early protein ICP27 induces DoTT through interaction with the mRNA 3’ processing factor CPSF and disruption of the processing complex.

    • Xiuye Wang
    • Thomas Hennig
    • Yongsheng Shi
    ResearchOpen Access
    Nature Communications
    Volume: 11, P: 1-13

  • Herpesvirus microRNAs interfere directly with host cell microRNA processing, thereby disrupting mitochondrial architecture, evading intrinsic host defences and driving the switch from latent to lytic infection.

    • Thomas Hennig
    • Archana B. Prusty
    • Bhupesh K. Prusty
    Research
    Nature
    Volume: 605, P: 539-544