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β-amyloid monomer scavenging by an anticalin protein prevents neuronal hyperactivity in mouse models of Alzheimer’s Disease

β-amyloid (Aβ)-dependent neuronal hyperactivity is an early marker of Alzheimer’s Disease (AD). Here, the authors report that scavenging Aβ monomers by an Aβ-binding anticalin protein blocks the formation of Aβ oligomers and prevents hyperactivity in AD mice.

Benedikt Zott
Lea Nästle
Arthur Konnerth
ResearchOpen Access10 Jul 2024
Nature Communications

Volume: 15, P: 1-13
Decreased amyloid-β and increased neuronal hyperactivity by immunotherapy in Alzheimer's models

Immunotherapy with antibodies targeting the amyloid-β peptide has yet to show any cognitive benefit in Alzheimer's disease patients in clinical trials. In vivo two-photon imaging in mouse models of Alzheimer's disease now reveals that these antibodies do not alleviate neuronal dysfunction and can even worsen it.

Marc Aurel Busche
Christine Grienberger
Arthur Konnerth
Research09 Nov 2015
Nature Neuroscience

Volume: 18, P: 1725-1727
Efficacy and safety of intracorneal allogenic ring segment implantation in keratoconus: 1-year results

Senay Asik Nacaroglu
Elif Ceren Yesilkaya
Aylin Kilic
Research16 Jun 2023
Eye

Volume: 37, P: 3807-3812
Predicting opioid consumption after surgical discharge: a multinational derivation and validation study using a foundation model

Chris Varghese
Luke Peters
Caroline Reinke
ResearchOpen Access26 Aug 2025
npj Digital Medicine

Volume: 8, P: 1-16

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