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Showing 1–4 of 4 results
Advanced filters: Author: Ben Elsworth Clear advanced filters
  • Observational studies have found overlap between Alzheimer’s disease and other diseases and phenotypes, although the causal relationships are unclear. Here, the authors perform an age-stratified phenome-wide association study of Alzheimer’s disease (AD) genetic liability and follow-up Mendelian randomization analyses to examine whether these phenotypes have a causal effect on AD.

    • Roxanna Korologou-Linden
    • Laxmi Bhatta
    • Neil M. Davies
    ResearchOpen Access
    Nature Communications
    Volume: 13, P: 1-14
  • Basal-like breast cancer is a heterogeneous disease with poor prognosis; however, its cellular origins and aetiology are poorly understood. Here the authors provide evidence that ID4 is a key controller of mammary stem/progenitor cell self-renewal, acting upstream of Notch signalling to repress luminal fate commitment.

    • Simon Junankar
    • Laura A. Baker
    • Alexander Swarbrick
    Research
    Nature Communications
    Volume: 6, P: 1-12
  • Family-based study designs have been applied to resolve confounding by population stratification, dynastic effects and assortative mating in genetic association analyses. Here, Brumpton et al. describe theory and simulations for overcoming such biases in Mendelian randomization through within-family studies.

    • Ben Brumpton
    • Eleanor Sanderson
    • Neil M. Davies
    ResearchOpen Access
    Nature Communications
    Volume: 11, P: 1-13
  • Bright star \(\nu\) Indi shows elevated levels of alpha-process elements, suggesting great age, and is kinematically heated, probably from the merger of a dwarf galaxy with the Milky Way. Chaplin et al. make a case for \(\nu\) Indi being an accurate indicator of the timing for the Gaia–Enceladus merger.

    • William J. Chaplin
    • Aldo M. Serenelli
    • Mutlu Yıldız
    Research
    Nature Astronomy
    Volume: 4, P: 382-389