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Showing 1–21 of 21 results

Advanced filters: Author: C Flotho Clear advanced filters

RAS mutations and clonality analysis in children with juvenile myelomonocytic leukemia (JMML)

C Flotho
S Valcamonica
CM Niemeyer
Research27 Jan 1999
Leukemia

Volume: 13, P: 32-37
Genome-wide single-nucleotide polymorphism analysis in juvenile myelomonocytic leukemia identifies uniparental disomy surrounding the NF1 locus in cases associated with neurofibromatosis but not in cases with mutant RAS or PTPN11

C Flotho
D Steinemann
C M Niemeyer
Research12 Mar 2007
Oncogene

Volume: 26, P: 5816-5821
Clinical evolution, genetic landscape and trajectories of clonal hematopoiesis in SAMD9/SAMD9L syndromes

This analysis of a large, clinically annotated cohort of individuals with predisposition to myelodysplastic syndromes reveals insights into the genetic determinants of disease progression and their relationship with clinical manifestations and therapy outcome.

Sushree S. Sahoo
Victor B. Pastor
Marcin W. Wlodarski
Research07 Oct 2021
Nature Medicine

Volume: 27, P: 1806-1817
RAS-pathway mutation patterns define epigenetic subclasses in juvenile myelomonocytic leukemia

Juvenile myelomonocytic leukemia (JMML) is an aggressive disease with limited options for treatment. Here, the authors analyse the DNA methylome and mutational profile of JMML to define three subgroups with unique molecular and clinical characteristics.

Daniel B. Lipka
Tania Witte
Christoph Plass
ResearchOpen Access19 Dec 2017
Nature Communications

Volume: 8, P: 1-14
Genome-wide DNA methylation is predictive of outcome in juvenile myelomonocytic leukemia

Juvenile myelomonocytic leukemia (JMML) is an aggressive disease with limited options for treatment. Here, the authors utilize DNA methylation based subgroups in JMML to predict clinical outcome.

Elliot Stieglitz
Tali Mazor
Mignon L. Loh
ResearchOpen Access19 Dec 2017
Nature Communications

Volume: 8, P: 1-8
Publisher Correction: Clinical evolution, genetic landscape and trajectories of clonal hematopoiesis in SAMD9/SAMD9L syndromes

Sushree S. Sahoo
Victor B. Pastor
Marcin W. Wlodarski
Amendments and Corrections19 Nov 2021
Nature Medicine

Volume: 27, P: 2248
Germline CBL mutations cause developmental abnormalities and predispose to juvenile myelomonocytic leukemia

Charlotte Niemeyer, Mignon Loh and colleagues report that germline mutations at CBL are associated with developmental abnormalities and predispose individuals to juvenile myelomonocytic leukemia.

Charlotte M Niemeyer
Michelle W Kang
Mignon L Loh
Research08 Aug 2010
Nature Genetics

Volume: 42, P: 794-800
Microarray-based classification of a consecutive series of 121 childhood acute leukemias: prediction of leukemic and genetic subtype as well as of minimal residual disease status

A Andersson
C Ritz
T Fioretos
Research05 Apr 2007
Leukemia

Volume: 21, P: 1198-1203
Signal transduction, cell cycle regulatory, and anti-apoptotic pathways regulated by IL-3 in hematopoietic cells: possible sites for intervention with anti-neoplastic drugs

WL Blalock
C Weinstein-Oppenheimer
JA McCubrey
Reviews09 Aug 1999
Leukemia

Volume: 13, P: 1109-1166
Autoimmunity and persistent RAS-mutated clones long after the spontaneous regression of JMML

M Takagi
J Piao
S Mizutani
Correspondence18 Mar 2013
Leukemia

Volume: 27, P: 1926-1928
Genomic and transcriptional landscape of P2RY8-CRLF2-positive childhood acute lymphoblastic leukemia

C Vesely
C Frech
R Panzer-Grümayer
ResearchOpen Access30 Nov 2016
Leukemia

Volume: 31, P: 1491-1501
Synonymous GATA2 mutations result in selective loss of mutated RNA and are common in patients with GATA2 deficiency

Emilia J. Kozyra
Victor B. Pastor
Marcin W. Wlodarski
ResearchOpen Access18 Jun 2020
Leukemia

Volume: 34, P: 2673-2687
Genotyping circulating tumor DNA of pediatric Hodgkin lymphoma

Ann-Kathrin Desch
Kristin Hartung
Andreas Bräuninger
Research20 Aug 2019
Leukemia

Volume: 34, P: 151-166
Successful double umbilical cord blood transplantation for relapsed juvenile myelomonocytic leukemia

C Flotho
T Vraetz
B Strahm
Correspondence24 Sept 2012
Leukemia

Volume: 27, P: 988-989
The DNA methyltransferase inhibitors azacitidine, decitabine and zebularine exert differential effects on cancer gene expression in acute myeloid leukemia cells

C Flotho
R Claus
M Lübbert
Research05 Feb 2009
Leukemia

Volume: 23, P: 1019-1028
Second allogeneic stem cell transplantation can rescue a significant proportion of patients with JMML relapsing after first allograft

Luca Vinci
Christian Flotho
Ayami Yoshimi
CorrespondenceOpen Access23 Feb 2023
Bone Marrow Transplantation

Volume: 58, P: 607-609
Randomized post-induction and delayed intensification therapy in high-risk pediatric acute lymphoblastic leukemia: long-term results of the international AIEOP-BFM ALL 2000 trial

Andishe Attarbaschi
Georg Mann
Martin Schrappe
Correspondence05 Dec 2019
Leukemia

Volume: 34, P: 1694-1700
Clonal duplication of a germline PTPN11 mutation due to acquired uniparental disomy in acute lymphoblastic leukemia blasts from a patient with Noonan syndrome

A Karow
D Steinemann
C P Kratz
Correspondence15 Mar 2007
Leukemia

Volume: 21, P: 1303-1305
Mutation analysis of Son of Sevenless in juvenile myelomonocytic leukemia

C P Kratz
C M Niemeyer
M Zenker
Correspondence22 Feb 2007
Leukemia

Volume: 21, P: 1108-1109
Classification of rare pediatric myeloid neoplasia—Quo vadis?

Charlotte M. Niemeyer
Martina Rudelius
Katherine R. Calvo
Correspondence21 Oct 2022
Leukemia

Volume: 36, P: 2947-2948
Mutational landscape in children with myelodysplastic syndromes is distinct from adults: specific somatic drivers and novel germline variants

V Pastor
S Hirabayashi
M W Wlodarski
Correspondence23 Nov 2016
Leukemia

Volume: 31, P: 759-762

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Search

RAS mutations and clonality analysis in children with juvenile myelomonocytic leukemia (JMML)

Genome-wide single-nucleotide polymorphism analysis in juvenile myelomonocytic leukemia identifies uniparental disomy surrounding the NF1 locus in cases associated with neurofibromatosis but not in cases with mutant RAS or PTPN11

Clinical evolution, genetic landscape and trajectories of clonal hematopoiesis in SAMD9/SAMD9L syndromes

RAS-pathway mutation patterns define epigenetic subclasses in juvenile myelomonocytic leukemia

Genome-wide DNA methylation is predictive of outcome in juvenile myelomonocytic leukemia

Publisher Correction: Clinical evolution, genetic landscape and trajectories of clonal hematopoiesis in SAMD9/SAMD9L syndromes

Germline CBL mutations cause developmental abnormalities and predispose to juvenile myelomonocytic leukemia

Microarray-based classification of a consecutive series of 121 childhood acute leukemias: prediction of leukemic and genetic subtype as well as of minimal residual disease status

Signal transduction, cell cycle regulatory, and anti-apoptotic pathways regulated by IL-3 in hematopoietic cells: possible sites for intervention with anti-neoplastic drugs

Autoimmunity and persistent RAS-mutated clones long after the spontaneous regression of JMML

Genomic and transcriptional landscape of P2RY8-CRLF2-positive childhood acute lymphoblastic leukemia

Synonymous GATA2 mutations result in selective loss of mutated RNA and are common in patients with GATA2 deficiency

Genotyping circulating tumor DNA of pediatric Hodgkin lymphoma

Successful double umbilical cord blood transplantation for relapsed juvenile myelomonocytic leukemia

The DNA methyltransferase inhibitors azacitidine, decitabine and zebularine exert differential effects on cancer gene expression in acute myeloid leukemia cells

Second allogeneic stem cell transplantation can rescue a significant proportion of patients with JMML relapsing after first allograft

Randomized post-induction and delayed intensification therapy in high-risk pediatric acute lymphoblastic leukemia: long-term results of the international AIEOP-BFM ALL 2000 trial

Clonal duplication of a germline PTPN11 mutation due to acquired uniparental disomy in acute lymphoblastic leukemia blasts from a patient with Noonan syndrome

Mutation analysis of Son of Sevenless in juvenile myelomonocytic leukemia

Classification of rare pediatric myeloid neoplasia—Quo vadis?

Mutational landscape in children with myelodysplastic syndromes is distinct from adults: specific somatic drivers and novel germline variants

Search

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