Thank you for visiting nature.com. You are using a browser version with limited support for CSS. To obtain the best experience, we recommend you use a more up to date browser (or turn off compatibility mode in Internet Explorer). In the meantime, to ensure continued support, we are displaying the site without styles and JavaScript.

View all journals
Search
Log in

nature
search

Search

Advanced search

Filter By:

Journal Check one or more journals to show results from those journals only.

Nature (4)
Scientific Reports (1)

Choose more journals

Article type Check one or more article types to show results from those article types only.

Research (4)
Amendments and Corrections (1)

Subject Check one or more subjects to show results from those subjects only.

Biochemistry
Cell biology
Diseases
Neuroscience

Date Choose a date option to show results from those dates only.

Today
Last 7 days
Last 30 days
Last 12 months
Last 2 years
Last 5 years

Custom date range

Clear all filters

Sort by:

Relevance

Date published (new to old)

Date published (old to new)

Showing 1–5 of 5 results

Advanced filters: Author: CHARLES G. GLABE Clear advanced filters

Retraction Note: A specific amyloid-β protein assembly in the brain impairs memory

Sylvain Lesné
Ming Teng Koh
Karen H. Ashe
Amendments and Corrections24 Jun 2024
Nature

Volume: 631, P: 240
Species specific agglutination of eggs by bindin isolated from sea urchin sperm

CHARLES G. GLABE
VICTOR D. VACQUIER
Research01 Jun 1977
Nature

Volume: 267, P: 836-838
RETRACTED ARTICLE: A specific amyloid-β protein assembly in the brain impairs memory

Mice of the type used to model Alzheimer's disease develop a hazy memory around middle age, well before they show neuronal loss and other disease-like symptoms — this has been attributed to extracellular accumulation of a soluble assembly of amyloid-β protein, dubbed Aβ star.

Sylvain Lesné
Ming Teng Koh
Karen H. Ashe
Research16 Mar 2006
Nature

Volume: 440, P: 352-357
Prion-like behaviour and tau-dependent cytotoxicity of pyroglutamylated amyloid-β

It is shown that the formation of amyloid-β oligomers, one of the histopathological signatures of Alzheimer’s disease, can be triggered by small quantities of a specifically truncated and post-translationally modified version of amyloid-β.

Justin M. Nussbaum
Stephan Schilling
George S. Bloom
Research02 May 2012
Nature

Volume: 485, P: 651-655
Intra- and extracellular β-amyloid overexpression via adeno-associated virus-mediated gene transfer impairs memory and synaptic plasticity in the hippocampus

Stefania Forner
Alessandra C. Martini
Frank M. LaFerla
ResearchOpen Access04 Nov 2019
Scientific Reports

Volume: 9, P: 1-11