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Communications Biology (1)

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Long QT syndrome type 3 gain-of-function of Na_v1.5 increases ventricular fibroblasts proliferation and pro-fibrotic factors

The Scn5a-ΔQKP mouse model of long QT syndrome type 3 unveils the role of Nav1.5 sodium channels expressed in cardiac fibroblasts and the contribution of those cells to pathophysiological processes involved in cardiac channelopathies.

Claire Castro
Justine Patin
Mickaël Derangeon
ResearchOpen Access11 Feb 2025
Communications Biology

Volume: 8, P: 1-12

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Long QT syndrome type 3 gain-of-function of Nav1.5 increases ventricular fibroblasts proliferation and pro-fibrotic factors

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Long QT syndrome type 3 gain-of-function of Na_v1.5 increases ventricular fibroblasts proliferation and pro-fibrotic factors