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Author Correction: Apoptotic stress-induced FGF signalling promotes non-cell autonomous resistance to cell death

Florian J. Bock
Egor Sedov
Stephen W. G. Tait
Amendments and CorrectionsOpen Access28 Feb 2024
Nature Communications

Volume: 15, P: 1
Leukaemia exposure alters the transcriptional profile and function of BCR::ABL1 negative macrophages in the bone marrow niche

The function of macrophages in myeloid leukaemia can be difficult to assess because of lack of differentiation between transformed and non-transformed cells. Here the authors use a chimeric mouse model to characterise the effect of myeloid leukaemia on bystander macrophages noting altered functional properties of these cells.

Amy Dawson
Martha M. Zarou
G. Vignir Helgason
ResearchOpen Access05 Feb 2024
Nature Communications

Volume: 15, P: 1-19
Apoptotic stress-induced FGF signalling promotes non-cell autonomous resistance to cell death

Apoptosis is a cellular process that eliminates damaged or superfluous cells. Here the authors show that cells undergoing apoptotic stresss secrete the growth factor FGF2, which upregulates pro-survival BCL-2 proteins in neighbouring cells, thereby promoting their survival.

Florian J. Bock
Egor Sedov
Stephen W. G. Tait
ResearchOpen Access12 Nov 2021
Nature Communications

Volume: 12, P: 1-14

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