Nature Search

ERCC1 and MUS81–EME1 promote sister chromatid separation by processing late replication intermediates at common fragile sites during mitosis

Breakage and rearrangement of common fragile sites (CFSs) can cause genomic instability. Rosselli and colleagues demonstrate that the endonuclease subunit ERCC1 and the endonuclease MUS18–EME1 are recruited to FANCD2-binding CFSs in mitosis to mediate their processing.

Valeria Naim
Therese Wilhelm
Filippo Rosselli
Research30 Jun 2013
Nature Cell Biology

Volume: 15, P: 1008-1015
The FANC pathway and BLM collaborate during mitosis to prevent micro-nucleation and chromosome abnormalities

The FANC and the BLM pathways collaborate during mitosis to prevent micronucleation and chromosome abnormalities.

Valeria Naim
Filippo Rosselli
Research24 May 2009
Nature Cell Biology

Volume: 11, P: 761-768
FANCA deficiency promotes leukaemic progression by allowing the emergence of cells carrying oncogenic driver mutations

Patrycja Pawlikowska
Laure Delestré
Christel Guillouf
ResearchOpen Access12 Aug 2023
Oncogene

Volume: 42, P: 2764-2775
FANCD2 functions as a critical factor downstream of MiTF to maintain the proliferation and survival of melanoma cells

Julie Bourseguin
Caroline Bonet
Corine Bertolotto
ResearchOpen Access09 Nov 2016
Scientific Reports

Volume: 6, P: 1-11
Fanconi anemia C gene product regulates expression of genes involved in differentiation and inflammation

Romina Zanier
Delphine Briot
Filippo Rosselli
Research12 Apr 2004
Oncogene

Volume: 23, P: 5004-5013
Fanconi anemia proteins counteract the implementation of the oncogene-induced senescence program

Anne Helbling-Leclerc
Françoise Dessarps-Freichey
Filippo Rosselli
ResearchOpen Access19 Nov 2019
Scientific Reports

Volume: 9, P: 1-11
V(D)J recombination process and the Pre-B to immature B-cells transition are altered in Fanca^−/− mice

Thuy Vy Nguyen
Patrycja Pawlikowska
Saïd Aoufouchi
ResearchOpen Access24 Nov 2016
Scientific Reports

Volume: 6, P: 1-11
Role of the ceramide-signaling pathways in ionizing radiation-induced apoptosis

Jean-Philippe Vit
Filippo Rosselli
Research27 Nov 2003
Oncogene

Volume: 22, P: 8645-8652
Large deletions in immunoglobulin genes are associated with a sustained absence of DNA Polymerase η

Leticia K. Lerner
Thuy V. Nguyen
Said Aoufouchi
ResearchOpen Access28 Jan 2020
Scientific Reports

Volume: 10, P: 1-11
Beyond DNA repair and chromosome instability—Fanconi anaemia as a cellular senescence-associated syndrome

Anne Helbling-Leclerc
Cécile Garcin
Filippo Rosselli
ReviewsOpen Access15 Mar 2021
Cell Death & Differentiation

Volume: 28, P: 1159-1173
NOX2-dependent ATM kinase activation dictates pro-inflammatory macrophage phenotype and improves effectiveness to radiation therapy

Qiuji Wu
Awatef Allouch
Jean-Luc Perfettini
Research02 Jun 2017
Cell Death & Differentiation

Volume: 24, P: 1632-1644
Werner's syndrome protein is phosphorylated in an ATR/ATM-dependent manner following replication arrest and DNA damage induced during the S phase of the cell cycle

Pietro Pichierri
Filippo Rosselli
Annapaola Franchitto
Research11 Mar 2003
Oncogene

Volume: 22, P: 1491-1500

Search

ERCC1 and MUS81–EME1 promote sister chromatid separation by processing late replication intermediates at common fragile sites during mitosis

The FANC pathway and BLM collaborate during mitosis to prevent micro-nucleation and chromosome abnormalities

FANCA deficiency promotes leukaemic progression by allowing the emergence of cells carrying oncogenic driver mutations

FANCD2 functions as a critical factor downstream of MiTF to maintain the proliferation and survival of melanoma cells

Fanconi anemia C gene product regulates expression of genes involved in differentiation and inflammation

Fanconi anemia proteins counteract the implementation of the oncogene-induced senescence program

V(D)J recombination process and the Pre-B to immature B-cells transition are altered in Fanca^−/− mice

Role of the ceramide-signaling pathways in ionizing radiation-induced apoptosis

Large deletions in immunoglobulin genes are associated with a sustained absence of DNA Polymerase η

Beyond DNA repair and chromosome instability—Fanconi anaemia as a cellular senescence-associated syndrome

NOX2-dependent ATM kinase activation dictates pro-inflammatory macrophage phenotype and improves effectiveness to radiation therapy

Werner's syndrome protein is phosphorylated in an ATR/ATM-dependent manner following replication arrest and DNA damage induced during the S phase of the cell cycle

Search

Quick links

Search

Filter By:

Search

Quick links