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Author Correction: Loss of ELF5–FBXW7 stabilizes IFNGR1 to promote the growth and metastasis of triple-negative breast cancer through interferon-γ signalling

Snahlata Singh
Sushil Kumar
Rumela Chakrabarti
Amendments and Corrections04 Aug 2021
Nature Cell Biology

Volume: 23, P: 1048
Loss of ELF5–FBXW7 stabilizes IFNGR1 to promote the growth and metastasis of triple-negative breast cancer through interferon-γ signalling

Singh et al. show that ELF5 loss disrupts IFNGR1 ubiquitination by FBXW7, thus enhancing IFN-γ signalling, neutrophil accumulation and PD-L1 expression to promote breast cancer progression and metastasis.

Snahlata Singh
Sushil Kumar
Rumela Chakrabarti
Research13 Apr 2020
Nature Cell Biology

Volume: 22, P: 591-602
Functional analysis of ESRP1/2 gene variants and CTNND1 isoforms in orofacial cleft pathogenesis

ESRP1, ESRP2 and CTNND1 operate in the periderm to regulate craniofacial morphogenesis. Using complementary in vivo and in vitro models, we tested the function of human ESRP1/2 gene variants and CTNND1 isoforms

Caroline Caetano da Silva
Claudio Macias Trevino
Eric C. Liao
ResearchOpen Access23 Aug 2024
Communications Biology

Volume: 7, P: 1-14

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Author Correction: Loss of ELF5–FBXW7 stabilizes IFNGR1 to promote the growth and metastasis of triple-negative breast cancer through interferon-γ signalling

Loss of ELF5–FBXW7 stabilizes IFNGR1 to promote the growth and metastasis of triple-negative breast cancer through interferon-γ signalling

Functional analysis of ESRP1/2 gene variants and CTNND1 isoforms in orofacial cleft pathogenesis

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