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Erratum

Hidemasa Goto
Tohru Kiyono
Masaki Inagaki
Amendments and Corrections01 Feb 2006
Nature Cell Biology

Volume: 8, P: 202
Protein kinases required for segregation of vimentin filaments in mitotic process

Yoshihiro Yasui
Hidemasa Goto
Masaki Inagaki
Research24 May 2001
Oncogene

Volume: 20, P: 2868-2876
Specific accumulation of Rho-associated kinase at the cleavage furrow during cytokinesis: cleavage furrow-specific phosphorylation of intermediate filaments

Hidetaka Kosako
Hidemasa Goto
Masaki Inagaki
Research28 Apr 1999
Oncogene

Volume: 18, P: 2783-2788
Complex formation of Plk1 and INCENP required for metaphase–anaphase transition

Hidemasa Goto
Tohru Kiyono
Masaki Inagaki
Research25 Dec 2005
Nature Cell Biology

Volume: 8, P: 180-187
PI 3-kinase-dependent phosphorylation of Plk1–Ser99 promotes association with 14-3-3γ and is required for metaphase–anaphase transition

Polo-like kinase 1 (Plk1) controls the transition between metaphase and anaphase during mitosis. Kasahara et al.show that Plk1 activity is regulated by phosphatidylinositide 3-kinase signalling through phosphorylation at a previously uncharacterized site.

Kousuke Kasahara
Hidemasa Goto
Masaki Inagaki
ResearchOpen Access21 May 2013
Nature Communications

Volume: 4, P: 1-12
Phosphorylation at serine 28 and acetylation at lysine 9 of histone H3 induced by trichostatin A

Shuping Zhong
Hidemasa Goto
Zigang Dong
Research14 Aug 2003
Oncogene

Volume: 22, P: 5291-5297
Direct interaction of Plk4 with STIL ensures formation of a single procentriole per parental centriole

Centrosome duplication requires a cartwheel-shaped protein complex containing the protein HsSAS-6, which acts as a template for centriole assembly. Ohta et al.show that HsSAS-6 is recruited to this structure by STIL and Plk4, and reveal that Plk4 phosphorylates STIL, stabilizing its interaction with HsSAS-6.

Midori Ohta
Tomoko Ashikawa
Daiju Kitagawa
ResearchOpen Access24 Oct 2014
Nature Communications

Volume: 5, P: 1-12
Production of a site- and phosphorylation state-specific antibody

Hidemasa Goto
Masaki Inagaki
Protocols18 Oct 2007
Nature Protocols

Volume: 2, P: 2574-2581
KRAS G12 mutations as adverse prognostic factors in KMT2A-rearranged acute myeloid leukemia

Shinju Iyoda
Kenichi Yoshida
Hidemasa Matsuo
Research18 Apr 2024
Leukemia

Volume: 38, P: 1609-1612
Time-saving method for directly amplifying and capturing a minimal amount of pancreatic tumor-derived mutations from fine-needle aspirates using digital PCR

Yusuke Ono
Akihiro Hayashi
Toshikatsu Okumura
ResearchOpen Access23 Jul 2020
Scientific Reports

Volume: 10, P: 1-10
Cdc7 kinase stimulates Aurora B kinase in M-phase

Sayuri Ito
Hidemasa Goto
Hisao Masai
ResearchOpen Access09 Dec 2019
Scientific Reports

Volume: 9, P: 1-15

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Erratum

Protein kinases required for segregation of vimentin filaments in mitotic process

Specific accumulation of Rho-associated kinase at the cleavage furrow during cytokinesis: cleavage furrow-specific phosphorylation of intermediate filaments

Complex formation of Plk1 and INCENP required for metaphase–anaphase transition

PI 3-kinase-dependent phosphorylation of Plk1–Ser99 promotes association with 14-3-3γ and is required for metaphase–anaphase transition

Phosphorylation at serine 28 and acetylation at lysine 9 of histone H3 induced by trichostatin A

Direct interaction of Plk4 with STIL ensures formation of a single procentriole per parental centriole

Production of a site- and phosphorylation state-specific antibody

KRAS G12 mutations as adverse prognostic factors in KMT2A-rearranged acute myeloid leukemia

Time-saving method for directly amplifying and capturing a minimal amount of pancreatic tumor-derived mutations from fine-needle aspirates using digital PCR

Cdc7 kinase stimulates Aurora B kinase in M-phase

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