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  • Mutagenic lesions such as those that give rise to cancer frequently segregate—unrepaired—during cell division, resulting in phasing of multiple alleles across generations of daughter cells and consequent tumour heterogeneity.

    • Sarah J. Aitken
    • Craig J. Anderson
    • Martin S. Taylor
    Research
    Nature
    Volume: 583, P: 265-270

  • How strand-asymmetric processes such as replication and transcription interact with DNA damage to drive mechanisms of repair and mutagenesis is explored.

    • Craig J. Anderson
    • Lana Talmane
    • Martin S. Taylor
    ResearchOpen Access
    Nature
    Volume: 630, P: 744-751

  • This Review provides a brief historical perspective of our understanding of the role of cancer genes before presenting the Integrative OncoGenomics (IntOGen) platform, a bioinformatics method of mutational driver identification, which is beginning to reveal the compendium of driver genes across many tumour types as well as alluding to their tumorigenic mechanisms.

    • Francisco Martínez-Jiménez
    • Ferran Muiños
    • Nuria Lopez-Bigas
    Reviews
    Nature Reviews Cancer
    Volume: 20, P: 555-572