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Showing 1–6 of 6 results
Advanced filters: Author: Inamul Kabir Clear advanced filters

  • Hepatocytes regenerate the liver after injury, however, the tissue repair mechanisms have been little explored. Here, the authors show that midlobular and pericentral hepatocytes increase their number and size in response to chemical, physical, and viral insults facilitating liver regeneration.

    • Inmaculada Ruz-Maldonado
    • John T. Gonzalez
    • Carlos Fernández-Hernando
    ResearchOpen Access
    Nature Communications
    Volume: 15, P: 1-23

  • Cellular senescence and smooth muscle cells are key features of the atherosclerotic plaque; however, how senescent cells regulate smooth muscle cells is largely unknown. Herein, a new study in Nature Aging illuminates this interplay, providing insights into plaque dynamics and stability with potentially profound implications for heart attack and stroke.

    • Inamul Kabir
    • Daniel M. Greif
    News & Views
    Nature Aging
    Volume: 1, P: 631-633

  • The pluripotency factor KLF4 has been described as pro-fibrotic or anti-fibrotic in various diseases. Herein, the authors show that during lung fibrosis, these distinct effects can be attributed to mesenchymal cell-type specific functions of KLF4.

    • Rachana R. Chandran
    • Yi Xie
    • Daniel M. Greif
    ResearchOpen Access
    Nature Communications
    Volume: 12, P: 1-17

  • Smooth muscle cells (SMCs) invade atherosclerotic lesions and expand, contributing to plaque progression. Here Misra et al. show that SMC-derived plaque cells come from a single SMC and integrin β3 in SMCs and macrophages regulate the fate, expansion and migration of SMCs during plaque formation.

    • Ashish Misra
    • Zhonghui Feng
    • Daniel M. Greif
    ResearchOpen Access
    Nature Communications
    Volume: 9, P: 1-13