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Erratum: Distinct Salmonella Enteritidis lineages associated with enterocolitis in high-income settings and invasive disease in low-income settings

Nicholas A Feasey
James Hadfield
Nicholas R Thomson
Amendments and Corrections30 Mar 2017
Nature Genetics

Volume: 49, P: 651
Phylogeographical analysis of the dominant multidrug-resistant H58 clade of Salmonella Typhi identifies inter- and intracontinental transmission events

Vanessa Wong and colleagues report whole-genome sequencing of 1,832 Salmonella enterica serovar Typhi isolates from 63 endemic countries. They identify mutations that define the multidrug resistant (MDR) H58 lineage and report numerous inter- and intracontinental transmissions of this lineage as well as an ongoing MDR typhoid epidemic in Africa.

Vanessa K Wong
Stephen Baker
Gordon Dougan
Research11 May 2015
Nature Genetics

Volume: 47, P: 632-639
Comparative genomics of Salmonella enterica serovar Enteritidis ST-11 isolated in Uruguay reveals lineages associated with particular epidemiological traits

Bruno D’Alessandro
Victoria Pérez Escanda
Laura Betancor
ResearchOpen Access27 Feb 2020
Scientific Reports

Volume: 10, P: 1-8
Distinct Salmonella Enteritidis lineages associated with enterocolitis in high-income settings and invasive disease in low-income settings

Nicholas Feasey and colleagues report whole-genome sequence analysis of 675 isolates of Salmonella enterica serovar Enteritidis from 45 countries. They find evidence for a global epidemic clade associated with enterocolitis and two novel clades restricted to distinct regions of Africa and associated with invasive disease.

Nicholas A Feasey
James Hadfield
Nicholas R Thomson
Research22 Aug 2016
Nature Genetics

Volume: 48, P: 1211-1217
An extended genotyping framework for Salmonella enterica serovar Typhi, the cause of human typhoid

Typhoid fever is caused by Salmonella enterica serovar Typhi (S. Typhi). This study examines ∼2,000 clinical isolates of S. Typhi to show highly structured/geographically restricted genomes except rapidly disseminating H58 subclade, and design a genotyping framework for tracking the disease.

Vanessa K. Wong
Stephen Baker
Ben Amos
ResearchOpen Access05 Oct 2016
Nature Communications

Volume: 7, P: 1-11
Publisher Correction: Comparative genomics of Salmonella enterica serovar Enteritidis ST-11 isolated in Uruguay reveals lineages associated with particular epidemiological traits

Bruno D’Alessandro
Victoria Pérez Escanda
Laura Betancor
Amendments and CorrectionsOpen Access13 May 2020
Scientific Reports

Volume: 10, P: 1-2
Enhancing colistin efficacy against Salmonella infections with a quinazoline-based dual therapeutic strategy

Carlos A. Lobertti
Fernán O. Gizzi
Eleonora García Véscovi
ResearchOpen Access01 Mar 2024
Scientific Reports

Volume: 14, P: 1-14
Quillaja brasiliensis saponin-based nanoparticulate adjuvants are capable of triggering early immune responses

Samuel Paulo Cibulski
Mariana Rivera-Patron
Fernando Silveira
ResearchOpen Access11 Sept 2018
Scientific Reports

Volume: 8, P: 1-15

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Erratum: Distinct Salmonella Enteritidis lineages associated with enterocolitis in high-income settings and invasive disease in low-income settings

Phylogeographical analysis of the dominant multidrug-resistant H58 clade of Salmonella Typhi identifies inter- and intracontinental transmission events

Comparative genomics of Salmonella enterica serovar Enteritidis ST-11 isolated in Uruguay reveals lineages associated with particular epidemiological traits

Distinct Salmonella Enteritidis lineages associated with enterocolitis in high-income settings and invasive disease in low-income settings

An extended genotyping framework for Salmonella enterica serovar Typhi, the cause of human typhoid

Publisher Correction: Comparative genomics of Salmonella enterica serovar Enteritidis ST-11 isolated in Uruguay reveals lineages associated with particular epidemiological traits

Enhancing colistin efficacy against Salmonella infections with a quinazoline-based dual therapeutic strategy

Quillaja brasiliensis saponin-based nanoparticulate adjuvants are capable of triggering early immune responses

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