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Advanced filters: Author: Joseph E Qualls Clear advanced filters

Toll-like receptor–induced arginase 1 in macrophages thwarts effective immunity against intracellular pathogens

Classically activated macrophages are targets of intracellular bacteria such as Mycobacteria tuberculosis. Murray and colleagues find that such pathogens induce arginase 1 in these macrophages to block the production of antibacterial nitric oxide.

Karim C El Kasmi
Joseph E Qualls
Peter J Murray
Research02 Nov 2008
Nature Immunology

Volume: 9, P: 1399-1406
A double agent in cancer: Stopping macrophages wounds tumors

Joseph E Qualls
Peter J Murray
Comments & Opinion01 Aug 2010
Nature Medicine

Volume: 16, P: 863-864
Immunosuppressive CD71⁺ erythroid cells compromise neonatal host defence against infection

In neonatal mice, susceptibility to infection is due to an enriched subset of arginase-2-expressing CD71⁺ erythroid cells, which suppresses the systemic activation of immune cells, thereby protecting neonates against aberrant inflammation triggered by colonization with commensal microbes.

Shokrollah Elahi
James M. Ertelt
Sing Sing Way
Research06 Nov 2013
Nature

Volume: 504, P: 158-162