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Publisher Correction: Non-functional ubiquitin C-terminal hydrolase L1 drives podocyte injury through impairing proteasomes in autoimmune glomerulonephritis

Julia Reichelt
Wiebke Sachs
Catherine Meyer-Schwesinger
Amendments and CorrectionsOpen Access28 Apr 2023
Nature Communications

Volume: 14, P: 1
Non-functional ubiquitin C-terminal hydrolase L1 drives podocyte injury through impairing proteasomes in autoimmune glomerulonephritis

In membranous nephropathy autoantibodies target podocytes of the kidney filter resulting in injury. Here the authors show that the ensuing proteostatic disturbances and proteinuria relate to aberrant interactions of non-functional UCH-L1 enzyme with the proteasome, curtailing its capacity.

Julia Reichelt
Wiebke Sachs
Catherine Meyer-Schwesinger
ResearchOpen Access13 Apr 2023
Nature Communications

Volume: 14, P: 1-19
Regeneration of the entire human epidermis using transgenic stem cells

Autologous transgenic epidermal stem cell cultures are used to reconstitute almost the entire epidermis of a patient with severe junctional epidermolysis bullosa.

Tobias Hirsch
Tobias Rothoeft
Michele De Luca
Research08 Nov 2017
Nature

Volume: 551, P: 327-332
Gene expression is stable in a complete CIB1 knockout keratinocyte model

Elias Imahorn
Magomet Aushev
Bettina Burger
ResearchOpen Access11 Sept 2020
Scientific Reports

Volume: 10, P: 1-9
Low-dose calcipotriol can elicit wound closure, anti-microbial, and anti-neoplastic effects in epidermolysis bullosa keratinocytes

Christina Guttmann-Gruber
Birgit Tockner
Josefina Piñón Hofbauer
ResearchOpen Access07 Sept 2018
Scientific Reports

Volume: 8, P: 1-14
Using pancreas tissue slices for in situ studies of islet of Langerhans and acinar cell biology

This protocol describes how to prepare mouse, rat, human and porcine pancreas tissue slices and how to use them to investigate cellular mechanisms underlying the function, pathology and interaction of endocrine and exocrine components of the pancreas.

Anja Marciniak
Christian M Cohrs
Stephan Speier
Protocols13 Nov 2014
Nature Protocols

Volume: 9, P: 2809-2822

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Publisher Correction: Non-functional ubiquitin C-terminal hydrolase L1 drives podocyte injury through impairing proteasomes in autoimmune glomerulonephritis

Non-functional ubiquitin C-terminal hydrolase L1 drives podocyte injury through impairing proteasomes in autoimmune glomerulonephritis

Regeneration of the entire human epidermis using transgenic stem cells

Gene expression is stable in a complete CIB1 knockout keratinocyte model

Low-dose calcipotriol can elicit wound closure, anti-microbial, and anti-neoplastic effects in epidermolysis bullosa keratinocytes

Using pancreas tissue slices for in situ studies of islet of Langerhans and acinar cell biology

Search

Quick links

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Publisher Correction: Non-functional ubiquitin C-terminal hydrolase L1 drives podocyte injury through impairing proteasomes in autoimmune glomerulonephritis

Non-functional ubiquitin C-terminal hydrolase L1 drives podocyte injury through impairing proteasomes in autoimmune glomerulonephritis

Regeneration of the entire human epidermis using transgenic stem cells

Gene expression is stable in a complete CIB1 knockout keratinocyte model

Low-dose calcipotriol can elicit wound closure, anti-microbial, and anti-neoplastic effects in epidermolysis bullosa keratinocytes

Using pancreas tissue slices for in situ studies of islet of Langerhans and acinar cell biology

Search

Quick links