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Showing 1–4 of 4 results

Advanced filters: Author: Kimiaki Katanosaka Clear advanced filters

TRPV2 mediates stress resilience in mouse cardiomyocytes

TRPV2-dependent signalling occurring in growing cardiomyocytes is essential for cardiac remodelling in response to haemodynamic loading in adults, indicating that TRPV2 is a mediator of stress resilience.

Yubing Dong
Guohao Wang
Yuki Katanosaka
ResearchOpen Access08 May 2025
Communications Biology

Volume: 8, P: 1-13
TRPV2 is critical for the maintenance of cardiac structure and function in mice

The TRPV2 calcium channel can be activated by mechanical stretch and may act as a mechanoreceptor in tissues. Here the authors deplete the TRPV2 calcium channel from the hearts of adult mice, showing that TRPV2 is important for the maintenance of cardiac structure and function.

Yuki Katanosaka
Keiichiro Iwasaki
Keiji Naruse
ResearchOpen Access29 May 2014
Nature Communications

Volume: 5, P: 1-14
TRPV2 is required for mechanical nociception and the stretch-evoked response of primary sensory neurons

Kimiaki Katanosaka
Satomi Takatsu
Yuki Katanosaka
ResearchOpen Access14 Nov 2018
Scientific Reports

Volume: 8, P: 1-10
Vacuolar-ATPase-mediated muscle acidification caused muscular mechanical nociceptive hypersensitivity after chronic stress in rats, which involved extracellular matrix proteoglycan and ASIC3

Teruaki Nasu
Amane Hori
Kazue Mizumura
ResearchOpen Access21 Aug 2023
Scientific Reports

Volume: 13, P: 1-17