Thank you for visiting nature.com. You are using a browser version with limited support for CSS. To obtain the best experience, we recommend you use a more up to date browser (or turn off compatibility mode in Internet Explorer). In the meantime, to ensure continued support, we are displaying the site without styles and JavaScript.

View all journals
Search
Log in

nature
search

Search

Advanced search

Filter By:

Journal Check one or more journals to show results from those journals only.

Molecular Psychiatry (2)
Nature Reviews Neurology (1)
Nature Reviews Neuroscience (1)
Nature Neuroscience (1)

Choose more journals

Article type Check one or more article types to show results from those article types only.

Research (2)
Reviews (2)
Amendments and Corrections (1)

Subject Check one or more subjects to show results from those subjects only.

Biological techniques
Diseases
Genetics
Neurology
Neuroscience

Date Choose a date option to show results from those dates only.

Today
Last 7 days
Last 30 days
Last 12 months
Last 2 years
Last 5 years

Custom date range

Clear all filters

Sort by:

Relevance

Date published (new to old)

Date published (old to new)

Showing 1–5 of 5 results

Advanced filters: Author: Liviu-Gabriel Bodea Clear advanced filters

LINE-1 retrotransposons contribute to mouse PV interneuron development

LINE-1 retrotransposons are a type of mobile DNA element normally repressed in the body. Here the authors show that LINE-1 sequences can jump in mouse parvalbumin interneurons and also promote the transcription of key parvalbumin interneuron genes.

Gabriela O. Bodea
Juan M. Botto
Geoffrey J. Faulkner
ResearchOpen Access21 May 2024
Nature Neuroscience

Volume: 27, P: 1274-1284
Correction: Scanning ultrasound-mediated memory and functional improvements do not require amyloid-β reduction

Gerhard Leinenga
Xuan Vinh To
Jürgen Götz
Amendments and CorrectionsOpen Access04 Jun 2024
Molecular Psychiatry

Volume: 29, P: 3968-3970
Rodent models for Alzheimer disease

Rodent models are extensively used to investigate the pathophysiology of Alzheimer disease. In this Review, Götz, Bodea and Goedert critically examine the approaches that have been adopted to generate rodent Alzheimer disease models and touch on some of the lessons that have been learned from their use.

Jürgen Götz
Liviu-Gabriel Bodea
Michel Goedert
Reviews07 Sept 2018
Nature Reviews Neuroscience

Volume: 19, P: 583-598
Amyloid-β and tau complexity — towards improved biomarkers and targeted therapies

Amyloid-β (Aβ) and tau come in a variety of forms and assembly states, not all of which are toxic. In this Review, Polanco and colleagues explain the clinical and therapeutic implications of this new understanding while highlighting the physiological and pathogenetic roles of Aβ and tau.

Juan Carlos Polanco
Chuanzhou Li
Jürgen Götz
Reviews15 Dec 2017
Nature Reviews Neurology

Volume: 14, P: 22-39
Scanning ultrasound-mediated memory and functional improvements do not require amyloid-β reduction

Gerhard Leinenga
Xuan Vinh To
Jürgen Götz
ResearchOpen Access18 Mar 2024
Molecular Psychiatry

Volume: 29, P: 2408-2423