Nature Search

Decontamination of Bacillus anthracis on Gruinard Island?

R. J. Manchee
M. G. Broster
J. Melling
Research19 May 1983
Nature

Volume: 303, P: 239-240
The orphan G protein-coupled receptor GPR55 promotes cancer cell proliferation via ERK

C Andradas
M M Caffarel
C Sánchez
Research06 Sept 2010
Oncogene

Volume: 30, P: 245-252
Bacillus anthracis on Gruinard Island

R. J. Manchee
M. G. Broster
A. J. Stagg
Research19 Nov 1981
Nature

Volume: 294, P: 254-255
Organic components in historical non-metallic seals identified using ¹³C-NMR spectroscopy

M. Cassar
G. V. Robins
A. Alstin
Research19 May 1983
Nature

Volume: 303, P: 238-239
The putative cannabinoid receptor GPR55 defines a novel autocrine loop in cancer cell proliferation

R Piñeiro
T Maffucci
M Falasca
Research13 Sept 2010
Oncogene

Volume: 30, P: 142-152
Nonlocal nonlinear phononics

Nonlinear phononics is a method for creating transient structural changes in solids, but its effect is limited to the region of optical excitation. Now, coupling to a propagating polariton allows nonlinear phononics to drive a nonlocal response.

M. Henstridge
M. Först
A. Cavalleri
ResearchOpen Access07 Mar 2022
Nature Physics

Volume: 18, P: 457-461
Anthrax island

R.J. MANCHEE
M.G. BROSTER
J. MELLING
Correspondence15 Apr 1982
Nature

Volume: 296, P: 598
Mitochondrial damage in muscle specific PolG mutant mice activates the integrated stress response and disrupts the mitochondrial folate cycle

Bond et al. show that inducible PolG mutation in muscle causes mtDNA damage and muscle wasting. This is driven by the integrated stress response (ISR) and reduction in folate intermediates, linking impaired folate metabolism with ISR/disease induction.

Simon T. Bond
Emily J. King
Brian G. Drew
ResearchOpen Access08 Mar 2025
Nature Communications

Volume: 16, P: 1-21
Uncoupling of the endocannabinoid signalling complex in a mouse model of fragile X syndrome

Fragile X syndrome is a major genetic cause of autism and is caused by loss of the fragile X mental retardation protein. In a mouse model of fragile X syndrome, Junget al. show that an absence of neuronal endocannabinoid signalling is responsible for the neurophysiological and behavioural defects.

Kwang-Mook Jung
Marja Sepers
Olivier J. Manzoni
ResearchOpen Access25 Sept 2012
Nature Communications

Volume: 3, P: 1-11
Anthrax island

M. STERNE
Correspondence04 Feb 1982
Nature

Volume: 295, P: 362
Correction: Corrigendum: 14-3-3ζ regulates the mitochondrial respiratory reserve linked to platelet phosphatidylserine exposure and procoagulant function

Simone M. Schoenwaelder
Roxane Darbousset
Shaun P. Jackson
Amendments and CorrectionsOpen Access30 Aug 2017
Nature Communications

Volume: 8, P: 1-2
Treatment of type 2 diabetes with the designer cytokine IC7Fc

The chimeric cytokine IC7Fc combines the beneficial effects of the cytokines IL-6 and CNTF on weight loss and metabolism in mice, with no obvious side effects in mice and non-human primates.

Maria Findeisen
Tamara L. Allen
Mark A. Febbraio
Research25 Sept 2019
Nature

Volume: 574, P: 63-68
Beyond the neuron–cellular interactions early in Alzheimer disease pathogenesis

Understanding the complex interplay of cells that protect neurons early in Alzheimer disease but later contribute to neurodegeneration is important for developing effective therapeutics. In this Review, Henstridge and colleagues discuss the contributions of multiple cell types to disease pathogenesis.

Christopher M. Henstridge
Bradley T. Hyman
Tara L. Spires-Jones
Reviews14 Jan 2019
Nature Reviews Neuroscience

Volume: 20, P: 94-108
Fructose stimulated de novo lipogenesis is promoted by inflammation

Fructose consumption has greatly increased in recent years and has been linked to the development of hepatic steatosis. Here, the authors show that fructose promotes gut-barrier deterioration and subsequent endotoxaemia that in turn induces hepatic lipogenesis by activation TLR signalling in liver macrophages.

Jelena Todoric
Giuseppe Di Caro
Michael Karin
Research24 Aug 2020
Nature Metabolism

Volume: 2, P: 1034-1045
Yap regulates skeletal muscle fatty acid oxidation and adiposity in metabolic disease

The mechanisms driving metabolic dysfunction in obesity remain incompletely understood. Here, the authors show that the levels of Hippo pathway effector YAP are reduced in muscle from individuals with insulin resistance and obese-diabetic mice, and that YAP promotes skeletal muscle lipid metabolism and limits adiposity in obese mice.

K. I. Watt
D. C. Henstridge
P. Gregorevic
ResearchOpen Access17 May 2021
Nature Communications

Volume: 12, P: 1-14
14-3-3ζ regulates the mitochondrial respiratory reserve linked to platelet phosphatidylserine exposure and procoagulant function

Platelets express negatively charged phosphatidylserine (PS) on their plasma membrane when propagating coagulation within a developing thrombus. Here the authors show that an adaptor protein 14-3-3 regulates mitochondrial function and PS exposure and thus platelet procoagulant activity, promising a new therapy to reduce thrombosis.

Simone M. Schoenwaelder
Roxane Darbousset
Shaun P. Jackson
ResearchOpen Access27 Sept 2016
Nature Communications

Volume: 7, P: 1-17
Cell-specific STORM super-resolution imaging reveals nanoscale organization of cannabinoid signaling

Combining patch-clamp recordings and subsequent STORM imaging of individual cells, the authors show that the axon terminals of perisomatically- and dendritically-projecting GABAergic interneurons show differences in CB₁ receptor number, active zone complexity, and receptor:effector ratio. Chronic exposure to THC evoked a dose-dependent and long-lasting downregulation of CB₁ at these synapses.

Barna Dudok
László Barna
István Katona
Research08 Dec 2014
Nature Neuroscience

Volume: 18, P: 75-86
Modeling Alzheimer’s disease brains in vitro

Alzheimer’s disease (AD) research is hampered by a lack of models that recapitulate all key disease features. Park et al. introduce a microfluidic device containing a 3D culture of human neurons, astrocytes, and microglia that develop AD-like pathology, revealing a potentially important inflammatory mechanism of neurodegeneration.

Christopher M. Henstridge
Tara L. Spires-Jones
News & Views27 Jun 2018
Nature Neuroscience

Volume: 21, P: 899-900
Hsp72 preserves muscle function and slows progression of severe muscular dystrophy

Increasing the expression of intramuscular heat shock protein 72 preserves muscle strength and ameliorates the dystrophic pathology in two mouse models of muscular dystrophy, suggesting a promising way forward for the treatment of muscular dystrophy.

Stefan M. Gehrig
Chris van der Poel
Gordon S. Lynch
Research04 Apr 2012
Nature

Volume: 484, P: 394-398
Class IIa HDACs inhibit cell death pathways and protect muscle integrity in response to lipotoxicity

Sheree D. Martin
Timothy Connor
Sean L. McGee
ResearchOpen Access01 Dec 2023
Cell Death & Disease

Volume: 14, P: 1-13

Search

Decontamination of Bacillus anthracis on Gruinard Island?

The orphan G protein-coupled receptor GPR55 promotes cancer cell proliferation via ERK

Bacillus anthracis on Gruinard Island

Organic components in historical non-metallic seals identified using ¹³C-NMR spectroscopy

The putative cannabinoid receptor GPR55 defines a novel autocrine loop in cancer cell proliferation

Nonlocal nonlinear phononics

Anthrax island

Mitochondrial damage in muscle specific PolG mutant mice activates the integrated stress response and disrupts the mitochondrial folate cycle

Uncoupling of the endocannabinoid signalling complex in a mouse model of fragile X syndrome

Anthrax island

Correction: Corrigendum: 14-3-3ζ regulates the mitochondrial respiratory reserve linked to platelet phosphatidylserine exposure and procoagulant function

Treatment of type 2 diabetes with the designer cytokine IC7Fc

Beyond the neuron–cellular interactions early in Alzheimer disease pathogenesis

Fructose stimulated de novo lipogenesis is promoted by inflammation

Yap regulates skeletal muscle fatty acid oxidation and adiposity in metabolic disease

14-3-3ζ regulates the mitochondrial respiratory reserve linked to platelet phosphatidylserine exposure and procoagulant function

Cell-specific STORM super-resolution imaging reveals nanoscale organization of cannabinoid signaling

Modeling Alzheimer’s disease brains in vitro

Hsp72 preserves muscle function and slows progression of severe muscular dystrophy

Class IIa HDACs inhibit cell death pathways and protect muscle integrity in response to lipotoxicity

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