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Showing 1–12 of 12 results
Advanced filters: Author: Maho Hamasaki Clear advanced filters

  • Maho Hamasaki is an associate professor at Osaka University, Japan. Maho’s laboratory focuses on investigating the mechanistic underpinnings of autophagy and the role of the autophagic process in disease.

    • Maho Hamasaki
    Comments & Opinion
    Nature Cell Biology
    Volume: 20, P: 1002

  • It was unknown how the most upstream Atg protein transits from the cytosol to autophagosome formation sites. Here, the authors show that ULK1 palmitoylation by ZDHHC13 recruits the complex to the formation site and enhances ATG14L phosphorylation.

    • Keisuke Tabata
    • Kenta Imai
    • Maho Hamasaki
    ResearchOpen Access
    Nature Communications
    Volume: 15, P: 1-15

  • The combination of light sheet illumination and reversibly switchable fluorophores enables improved structured illumination microscopy for fast, low-background super-resolution imaging in cells and spheroids.

    • Kenta Temma
    • Ryosuke Oketani
    • Katsumasa Fujita
    Research
    Nature Methods
    Volume: 21, P: 889-896

  • Yanagawa et al. show that the autophagy-related protein Rubicon recruits WIPI2d to endosomes to promote exosome biogenesis. Rubicon promotes both an increase in exosome release during ageing and the pro-senescent effects of these exosomes.

    • Kyosuke Yanagawa
    • Akiko Kuma
    • Tamotsu Yoshimori
    Research
    Nature Cell Biology
    Volume: 26, P: 1558-1570

  • Selective autophagy engages several cargo receptors that target specific, potentially toxic, content (damaged organelles, protein aggregates, pathogens) for lysosomal degradation. Understanding of the mechanisms governing this process in mammals has expanded in recent years, opening the prospects for enhancing selective autophagy to boost cellular quality control capabilities.

    • Jose Norberto S. Vargas
    • Maho Hamasaki
    • Tamotsu Yoshimori
    Reviews
    Nature Reviews Molecular Cell Biology
    Volume: 24, P: 167-185

  • Scientific questions are universal but the scientific workforce remains skewed, with women and gender minorities still underrepresented. Initiatives such as the Women in Autophagy network promote the careers of these underrepresented groups with a range of free, year-round scientific, mentoring and networking activities for all scientists.

    • Mericka McCabe
    • Patricia Boya
    • Ana Maria Cuervo
    Comments & Opinion
    Nature Cell Biology
    Volume: 26, P: 2009-2012

  • Autophagic activity decreases with age via unknown mechanisms. Here the authors show that expression of the negative autophagy regulator Rubicon increases with age, that its genetic ablation improves lifespan and ameliorates a number of age-associated phenotypes in invertebrates and in mouse models.

    • Shuhei Nakamura
    • Masaki Oba
    • Tamotsu Yoshimori
    ResearchOpen Access
    Nature Communications
    Volume: 10, P: 1-11

  • Autophagic activity declines with age in several tissues and is linked to aging-associated functional decline and pathologies. Here the authors show that Rubicon, a negative regulator of autophagy, decreases in adipocytes with age, and its loss leads to adipocyte dysfunction via excess autophagic degradation of SRC-1 and TIF2.

    • Tadashi Yamamuro
    • Tsuyoshi Kawabata
    • Tamotsu Yoshimori
    ResearchOpen Access
    Nature Communications
    Volume: 11, P: 1-16

  • This study shows that autophagosomes form at sites of contact between the endoplasmic reticulum and mitochondria, and that formation requires the SNARE protein syntaxin 17.

    • Maho Hamasaki
    • Nobumichi Furuta
    • Tamotsu Yoshimori
    Research
    Nature
    Volume: 495, P: 389-393