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Elevated CSF GAP-43 is associated with accelerated tau accumulation and spread in Alzheimer’s disease

Trans-synaptic tau spread drives neurodegeneration in Alzheimer’s disease. This study shows that GAP-43, a marker of synaptic abnormality, is linked to faster tau spread, showing that synaptic changes may contribute to tau spreading in Alzheimer’s disease.

Nicolai Franzmeier
Amir Dehsarvi
Michael Schöll
ResearchOpen Access03 Jan 2024
Nature Communications

Volume: 15, P: 1-10

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Elevated CSF GAP-43 is associated with accelerated tau accumulation and spread in Alzheimer’s disease

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