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Showing 1–5 of 5 results

Advanced filters: Author: Neta Agmon Clear advanced filters

Author Correction: A scalable peptide-GPCR language for engineering multicellular communication

Sonja Billerbeck
James Brisbois
Virginia W. Cornish
Amendments and CorrectionsOpen Access29 Jan 2019
Nature Communications

Volume: 10, P: 1
A scalable peptide-GPCR language for engineering multicellular communication

A bottleneck to engineered multicellular systems is the lack of a scalable signalling language. Here the authors use fungal mating peptide and GPCR pairs to build functional cell-to-cell communication links.

Sonja Billerbeck
James Brisbois
Virginia W. Cornish
ResearchOpen Access29 Nov 2018
Nature Communications

Volume: 9, P: 1-12
Engineered dual selection for directed evolution of SpCas9 PAM specificity

The PAM specificity of SpCas9 can be altered with positive selection during directed evolution. Here the authors use simultaneous positive and negative selection to improve activity on NAG PAMs while reducing activity on NGG PAMs.

Gregory W. Goldberg
Jeffrey M. Spencer
Marcus B. Noyes
ResearchOpen Access13 Jan 2021
Nature Communications

Volume: 12, P: 1-16
BRCA1 and S phase DNA repair pathways restrict LINE-1 retrotransposition in human cells

Whole-genome siRNA screens to identify regulators of human LINE-1 retrotransposition reveal that BRCA1 and Fanconi anemia DNA repair factors inhibit retrotransposition at stalled replication fork targets created and exploited by L1.

Paolo Mita
Xiaoji Sun
Jef D. Boeke
Research10 Feb 2020
Nature Structural & Molecular Biology

Volume: 27, P: 179-191
Effect of nuclear architecture on the efficiency of double-strand break repair

For double-strand breaks to be repaired by homologous recombination, the homologous template must be in the nuclear space. By elegant genetic manipulation of yeast chromosomes, Kupiec and colleagues show that nuclear organization influences repair efficiency.

Neta Agmon
Batia Liefshitz
Martin Kupiec
Research05 May 2013
Nature Cell Biology

Volume: 15, P: 694-699