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Showing 1–5 of 5 results

Advanced filters: Author: Noa Lamm Clear advanced filters

Nuclear F-actin counteracts nuclear deformation and promotes fork repair during replication stress

Lamm et al. report that replication stress activates mTOR through ATR to induce nuclear actin polymerization, facilitating the recovery from replication stress.

Noa Lamm
Mark N. Read
Anthony J. Cesare
Research30 Nov 2020
Nature Cell Biology

Volume: 22, P: 1460-1470
A CPC-shelterin-BTR axis regulates mitotic telomere deprotection

Here the authors reveal how telomeres signal mitotic stress. A key protein network alters their structure exposing telomere ends to signal mitotic stress, ultimately triggering a controlled DNA damage response to remove faulty cells.

Diana Romero-Zamora
Samuel Rogers
Anthony J. Cesare
ResearchOpen Access17 Mar 2025
Nature Communications

Volume: 16, P: 1-19
ZNF827 is a single-stranded DNA binding protein that regulates the ATR-CHK1 DNA damage response pathway

Here, the authors characterise the zinc finger protein ZNF827 as a single stranded DNA binding protein that accumulates at stalled replication forks to activate the ATR-CHK1 pathway and engage homologous-recombination mediated DNA repair.

Sile F. Yang
Christopher B. Nelson
Hilda A. Pickett
ResearchOpen Access12 Mar 2024
Nature Communications

Volume: 15, P: 1-15
Replication stress induces mitotic death through parallel pathways regulated by WAPL and telomere deprotection

Mitotic catastrophe is a regulated mechanism that responds to aberrant mitoses leading to removal of damaged cells. Here the authors reveal how replication stress induces mitotic death through pathways regulated by WAPL and telomere deprotection.

V. Pragathi Masamsetti
Ronnie Ren Jie Low
Anthony J. Cesare
ResearchOpen Access17 Sept 2019
Nature Communications

Volume: 10, P: 1-15
The International Climate Psychology Collaboration: Climate change-related data collected from 63 countries

Kimberly C. Doell
Boryana Todorova
Jay J. Van Bavel
ResearchOpen Access01 Oct 2024
Scientific Data

Volume: 11, P: 1-17