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Showing 1–11 of 11 results

Advanced filters: Author: Ruth M. Stassart Clear advanced filters

Publisher Correction: NRG1 type I dependent autoparacrine stimulation of Schwann cells in onion bulbs of peripheral neuropathies

Robert Fledrich
Dagmar Akkermann
Ruth M. Stassart
Amendments and CorrectionsOpen Access16 Apr 2019
Nature Communications

Volume: 10, P: 1-2
Multi-omic identification of perineurial hyperplasia and lipid-associated nerve macrophages in human polyneuropathies

Polyneuropathy causes are difficult to pinpoint. Here, authors used single-nucleus and spatial transcriptomics of 37 human peripheral nerves to identify focal perineurial hyperplasia and endoneurial lipid-associated macrophages in polyneuropathies.

Michael Heming
Anna-Lena Börsch
Gerd Meyer zu Hörste
ResearchOpen Access23 Aug 2025
Nature Communications

Volume: 16, P: 1-14
Addendum: Myelin insulation as a risk factor for axonal degeneration in autoimmune demyelinating disease

Erik Schäffner
Mar Bosch-Queralt
Ruth M. Stassart
Amendments and CorrectionsOpen Access11 Mar 2025
Nature Neuroscience

Volume: 28, P: 914
Myelin insulation as a risk factor for axonal degeneration in autoimmune demyelinating disease

Myelin is widely believed to protect axons from damage in multiple sclerosis. The authors challenge this view and find that myelin insulation renders axons more vulnerable to degeneration in an autoimmune environment.

Erik Schäffner
Mar Bosch-Queralt
Ruth M. Stassart
ResearchOpen Access29 Jun 2023
Nature Neuroscience

Volume: 26, P: 1218-1228
A role for Schwann cell–derived neuregulin-1 in remyelination

The authors show that nerve injury induces expression of NRG1 type I in Schwann cells and that this expression is necessary for efficient remyelination. In addition, axonally expressed NRG1 type III can negatively regulate the expression of NRG1 type I in Schwann cells.

Ruth M Stassart
Robert Fledrich
Klaus-Armin Nave
Research09 Dec 2012
Nature Neuroscience

Volume: 16, P: 48-54
Hypoxia-induced conversion of sensory Schwann cells into repair cells is regulated by HDAC8

HDAC8 counteracts the conversion of sensory Schwann cells into their repair phenotype by controlling hypoxia-induced c-Jun upregulation after injury. Ablating HDAC8 accelerates the regrowth of sensory axons and the recovery of the sensory function.

Nadège Hertzog
Mert Duman
Claire Jacob
ResearchOpen Access09 Jan 2025
Nature Communications

Volume: 16, P: 1-16
NRG1 type I dependent autoparacrine stimulation of Schwann cells in onion bulbs of peripheral neuropathies

Onion bulbs are a hallmark of demyelinating peripheral neuropathies. Here the authors identify Neuregulin-1 type I expression in Schwann cells as an essential mechanism involved in the formation of these characteristic structures.

Robert Fledrich
Dagmar Akkermann
Ruth M. Stassart
ResearchOpen Access01 Apr 2019
Nature Communications

Volume: 10, P: 1-17
Soluble neuregulin-1 modulates disease pathogenesis in rodent models of Charcot-Marie-Tooth disease 1A

Postnatal administration of neuregulin-1 into rodent models of the peripheral neuropathy Charcot-Marie-Tooth disease can ameliorate motor symptoms.

Robert Fledrich
Ruth M Stassart
Michael W Sereda
Research24 Aug 2014
Nature Medicine

Volume: 20, P: 1055-1061
Myelin dysfunction drives amyloid-β deposition in models of Alzheimer’s disease

Mouse models show that myelin dysfunction and associated inflammation increase with age, which can promote amyloid-β deposition and therefore risk of developing Alzheimer’s disease.

Constanze Depp
Ting Sun
Klaus-Armin Nave
ResearchOpen Access31 May 2023
Nature

Volume: 618, P: 349-357
Dietary cholesterol promotes repair of demyelinated lesions in the adult brain

Cholesterol is important for axonal myelination during development. Here the authors show that cholesterol levels are reduced in a cuprizone mouse model of multiple sclerosis and that dietary cholesterol supplementation enhances remyelination and recovery.

Stefan A. Berghoff
Nina Gerndt
Gesine Saher
ResearchOpen Access24 Jan 2017
Nature Communications

Volume: 8, P: 1-15
Survival-time dependent increase in neuronal IL-6 and astroglial GFAP expression in fatally injured human brain tissue

Florian Trautz
Heike Franke
Benjamin Ondruschka
ResearchOpen Access15 Aug 2019
Scientific Reports

Volume: 9, P: 1-15