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Showing 1–5 of 5 results
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  • A suppressive type of immune cell called a regulatory T cell has a key role in helping muscles to adapt to exercise — guarding muscle mitochondrial organelles against damage mediated by proinflammatory factors generated during physical activity.

    • Gerald Coulis
    • S. Armando Villalta
    News & Views
    Nature
    Volume: 625, P: 35-36

  • A remote, downstream event in the pathology of muscular dystrophy may have a key role in the disease (pages 325–330). It seems that induction of nitric oxide synthase causes calcium to leak inside the cell through ryanodine receptors. The findings provide new options for therapeutic interventions.

    • James G Tidball
    • S Armando Villalta
    News & Views
    Nature Medicine
    Volume: 15, P: 243-244

  • Circulating monocytes contribute to the transition to pain chronicity but the molecular events that cause their deployment are still unclear. Using a mouse model of hyperalgesic priming, here the authors show that blood monocytes contribute to the emergence of chronic pain via a mechanism that requires a transient disruption of NAAA-regulated lipid signaling.

    • Yannick Fotio
    • Alex Mabou Tagne
    • Daniele Piomelli
    ResearchOpen Access
    Nature Communications
    Volume: 15, P: 1-15

  • Mohammadi et al. show that the transplantation of pancreatic islets within mesenchymal-stem cell (MSC) exosome-loaded hybrid alginate microcapsules in diabetic mice lead to diminished immune-based foreign body response and hyperglycemia control. This is due to immunomodulatory behavior of MSC exosomes at the engraftment site where they exert local immunosuppressive effects.

    • M. Rezaa Mohammadi
    • Samuel Mathew Rodriguez
    • Jonathan R. T. Lakey
    ResearchOpen Access
    Communications Biology
    Volume: 4, P: 1-17