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Presence of gene for β globin in homozygous β₀ thalassaemia

Paul Tolstoshev
Joan Mitchell
J. B. Clegg
Research15 Jan 1976
Nature

Volume: 259, P: 95-98
The deletion in a type of δ⁰-β⁰-thalassaemia begins in an inverted AluI repeat

Sergio Ottolenghi
Barbara Giglioni
Research23 Dec 1982
Nature

Volume: 300, P: 770-771
Gene deletion as the cause of α thalassaemia: The severe form of α thalassaemia is caused by a haemoglobin gene deletion

SERGIO OTTOLENGHI
W. GEORGE LANYON
WONG HOCK BOON
Research04 Oct 1974
Nature

Volume: 251, P: 389-392
Polymorphisms of human γ-globin genes in Mediterranean populations

Peter F. R. Little
Robert Williamson
Umberto Mazza
Research15 Nov 1979
Nature

Volume: 282, P: 316-318
Hippocampal development and neural stem cell maintenance require Sox2-dependent regulation of Shh

Deleting the transcription factor SOX2 in mouse embryonic brain causes a loss of neural stem cells and neurogenesis in the hippocampus. An SHH pharmacological agonist partially rescues the hippocampal defect and SHH was found to be a SOX2 target.

Rebecca Favaro
Menella Valotta
Silvia K Nicolis
Research06 Sept 2009
Nature Neuroscience

Volume: 12, P: 1248-1256

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Presence of gene for β globin in homozygous β₀ thalassaemia

The deletion in a type of δ⁰-β⁰-thalassaemia begins in an inverted AluI repeat

Gene deletion as the cause of α thalassaemia: The severe form of α thalassaemia is caused by a haemoglobin gene deletion

Polymorphisms of human γ-globin genes in Mediterranean populations

Hippocampal development and neural stem cell maintenance require Sox2-dependent regulation of Shh

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Presence of gene for β globin in homozygous β0 thalassaemia

The deletion in a type of δ0-β0-thalassaemia begins in an inverted AluI repeat

Gene deletion as the cause of α thalassaemia: The severe form of α thalassaemia is caused by a haemoglobin gene deletion

Polymorphisms of human γ-globin genes in Mediterranean populations

Hippocampal development and neural stem cell maintenance require Sox2-dependent regulation of Shh

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Quick links

Presence of gene for β globin in homozygous β₀ thalassaemia

The deletion in a type of δ⁰-β⁰-thalassaemia begins in an inverted AluI repeat