This study reports the viral and cellular N
6-methyladenosine (m6A) and N
6,2′-O-dimethyladenosine (m6Am) epitranscriptomes during KSHV latent and lytic infection, and shows that lytic replication induces dynamic epitranscriptome reprogramming of host pathways that control this process.
- Brandon Tan
- Hui Liu
- Shou-Jiang Gao
