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Galectin-3 causes enteric neuronal loss in mice after left sided permanent middle cerebral artery occlusion, a model of stroke

Xiaowen Cheng
Antonio Boza-Serrano
Ulrikke Voss
ResearchOpen Access09 Sept 2016
Scientific Reports

Volume: 6, P: 1-8
‘Proactive aging’ is a new research approach for a new era

Susanne Iwarsson
Håkan Jönson
Martin Englund
Correspondence08 Jun 2023
Nature Aging

Volume: 3, P: 755-756
Galectin-3 released in response to traumatic brain injury acts as an alarmin orchestrating brain immune response and promoting neurodegeneration

Ping Kei Yip
Alejandro Carrillo-Jimenez
Miguel Angel Burguillos
ResearchOpen Access27 Jan 2017
Scientific Reports

Volume: 7, P: 1-13
Soluble cerebral Aβ protofibrils link Aβ plaque pathology to changes in CSF Aβ₄₂/Aβ₄₀ ratios, neurofilament light and tau in Alzheimer’s disease model mice

Andersson et al. identify that high Aβ₄₂/Aβ₄₀ ratios in brain protofibrils best predict lower Aβ₄₂/Aβ₄₀ ratios in the cerebrospinal fluid (CSF) and higher neurofilament light/total tau levels, indicating that CSF changes in Alzheimer’s disease reflect soluble protofibrils more accurately than amyloid plaques.

Emelie Andersson
Nils Lindblom
Oskar Hansson
ResearchOpen Access12 Feb 2025
Nature Aging

Volume: 5, P: 366-375
Caspase signalling controls microglia activation and neurotoxicity

Miguel A. Burguillos
Tomas Deierborg
Bertrand Joseph
Research09 Mar 2011
Nature

Volume: 472, P: 319-324
The effect of electroconvulsive therapy on neuroinflammation, behavior and amyloid plaques in the 5xFAD mouse model of Alzheimer’s disease

Martina Svensson
Gustaf Olsson
Tomas Deierborg
ResearchOpen Access01 Mar 2021
Scientific Reports

Volume: 11, P: 1-11
Voluntary running does not reduce neuroinflammation or improve non-cognitive behavior in the 5xFAD mouse model of Alzheimer’s disease

Martina Svensson
Emelie Andersson
Tomas Deierborg
ResearchOpen Access28 Jan 2020
Scientific Reports

Volume: 10, P: 1-10
Innate immune alterations are elicited in microglial cells before plaque deposition in the Alzheimer’s disease mouse model 5xFAD

Antonio Boza-Serrano
Yiyi Yang
Tomas Deierborg
ResearchOpen Access24 Jan 2018
Scientific Reports

Volume: 8, P: 1-16
Dopaminergic neurons lacking Caspase-3 avoid apoptosis but undergo necrosis after MPTP treatment inducing a Galectin-3-dependent selective microglial phagocytic response

Juan García-Revilla
Rocío Ruiz
José Luis Venero
ResearchOpen Access27 Aug 2024
Cell Death & Disease

Volume: 15, P: 1-17
Correlative optical photothermal infrared and X-ray fluorescence for chemical imaging of trace elements and relevant molecular structures directly in neurons

A new imaging approach that combines two techniques standing at the opposite ends of the electromagnetic spectrum.

Nadja Gustavsson
Agnes Paulus
Oxana Klementieva
ResearchOpen Access22 Jul 2021
Light: Science & Applications

Volume: 10, P: 1-10
FGF family members differentially regulate maturation and proliferation of stem cell-derived astrocytes

Ekaterina Savchenko
Gabriel N. Teku
Laurent Roybon
ResearchOpen Access03 Jul 2019
Scientific Reports

Volume: 9, P: 1-13
Potent pro-inflammatory and pro-fibrotic molecules, osteopontin and galectin-3, are not major disease modulators of laminin α2 chain-deficient muscular dystrophy

Kinga I. Gawlik
Johan Holmberg
Madeleine Durbeej
ResearchOpen Access10 Mar 2017
Scientific Reports

Volume: 7, P: 1-18
Conditional ablation of myeloid TNF increases lesion volume after experimental stroke in mice, possibly via altered ERK1/2 signaling

Bettina Hjelm Clausen
Matilda Degn
Kate Lykke Lambertsen
ResearchOpen Access07 Jul 2016
Scientific Reports

Volume: 6, P: 1-16
Galectin-3, a rising star in modulating microglia activation under conditions of neurodegeneration

Juan García-Revilla
Antonio Boza-Serrano
Jose L. Venero
ReviewsOpen Access20 Jul 2022
Cell Death & Disease

Volume: 13, P: 1-11

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