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Showing 1–8 of 8 results

Advanced filters: Author: Umesh K Bhanot Clear advanced filters

SMAD4 induces opposite effects on metastatic growth from pancreatic tumors depending on the organ of residence

Using mouse pancreatic cancer models, Tsanov et al. show that reactivation of SMAD4 at metastatic sites promotes tumor growth in the lung through RUNX1 but restrains metastatic growth through KLF4 in the liver, influenced by organ-specific epigenetic states.

Kaloyan M. Tsanov
Francisco M. Barriga
Scott W. Lowe
ResearchOpen Access25 Sept 2025
Nature Cancer

P: 1-18
FOXA2 promotes metastatic competence in small cell lung cancer

Small cell lung cancer presents high and multi-site metastatic potential. Here, the authors discover that FOXA2, regulated by ASCL1, promotes multi-site metastasis in small cell lung cancer by inducing a fetal neuroendocrine gene expression program.

Kenta Kawasaki
Sohrab Salehi
Charles M. Rudin
ResearchOpen Access26 May 2025
Nature Communications

Volume: 16, P: 1-16
ILC2s amplify PD-1 blockade by activating tissue-specific cancer immunity

Tumour-infiltrating group 2 innate lymphoid cells prime CD8⁺ T cells and amplify the anti-tumour effects of PD-1 blockade in pancreatic ductal adenocarcinoma.

John Alec Moral
Joanne Leung
Vinod P. Balachandran
Research19 Feb 2020
Nature

Volume: 579, P: 130-135
Identification of unique neoantigen qualities in long-term survivors of pancreatic cancer

The analysis of T-cell antigens in long-term survivors of pancreatic ductal adenocarcinoma suggests that neoantigen immunogenicity and quality, not purely quantity, correlate with survival.

Vinod P. Balachandran
Marta Łuksza
Steven D. Leach
Research01 Nov 2017
Nature

Volume: 551, P: 512-516
CDC7 inhibition impairs neuroendocrine transformation in lung and prostate tumors through MYC degradation

Alvaro Quintanal-Villalonga
Kenta Kawasaki
Charles M. Rudin
ResearchOpen Access26 Jul 2024
Signal Transduction and Targeted Therapy

Volume: 9, P: 1-14
Parenchymal regression in chronic pancreatitis spares islets reprogrammed for the expression of NFκB and IAPs

Cornelia Hasel
Umesh K Bhanot
Peter Möller
Research22 Aug 2005
Laboratory Investigation

Volume: 85, P: 1263-1275
Mechanisms of parenchymal injury and signaling pathways in ectatic ducts of chronic pancreatitis: implications for pancreatic carcinogenesis

Umesh K Bhanot
Peter Möller
Research23 Mar 2009
Laboratory Investigation

Volume: 89, P: 489-497
Pancreatic intraductal tubulopapillary neoplasm is genetically distinct from intraductal papillary mucinous neoplasm and ductal adenocarcinoma

Olca Basturk
Michael F Berger
David S Klimstra
Research04 Aug 2017
Modern Pathology

Volume: 30, P: 1760-1772