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Acknowledgements
We thank Drs P Bouillet and M Lavin for gifts of mice; C Gatt, S O’Connor, J Mansheim, K McKenzie and G Siciliano for expert animal care; B Helbert for genotyping; J Corbin and J McManus for automated blood analysis. This work was supported by grants and fellowships from the Cancer Council of Victoria (SG, AD ‘Sydney Parker Smith Postdoctoral Fellowship’), Leukaemia Foundation Australia (SG), the Lady Tata Memorial Trust (SG), Cure Brain Cancer Australia (AS), the National Health and Medical Research Council (Program Grants #1016701 and 1016647, NHMRC Australia Fellowship 1020363 to AS), the Leukemia and Lymphoma Society (SOCR Grant #7001-03 to AS), Melbourne International Research and the Melbourne International Fee Remission Scholarship (University of Melbourne, SG) and Cancer Therapeutics CRC Top-up Scholarship (SG, AD). The estate of Anthony (Toni) Redstone OAM, University of Melbourne International Research and International Fee Remission Scholarships (SG), Australian Postgraduate Award (ARDD), and the operational infrastructure grants through the Australian Government IRIISS and the Victorian State Government OIS.
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Delbridge, A., Grabow, S. & Strasser, A. Loss of BIM augments resistance of ATM-deficient thymocytes to DNA damage-induced apoptosis but does not accelerate lymphoma development. Cell Death Differ 24, 1987–1988 (2017). https://doi.org/10.1038/cdd.2017.138
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DOI: https://doi.org/10.1038/cdd.2017.138
