Fig. 2: Aspirin antiplatelet effect pathway.

Aspirin is an inhibitor of cyclooxygenase (COX) that catalyses the transformation of arachidonic acid (AA) into prostaglandins G2 and H2. Thromboxane A2 (TXA2) is formed by thromboxane synthase and binds to the TXA2 receptors of nearby platelets, thereby serving as a potent platelet activator (modified from ref. 69). Figure created with Bio Render.com.