Fig. 10

Ds effectively attenuated nano-COM-induced HK-2 damage and delayed the EMT process by interfering with TGF-β expression and secretion and inhibiting the activation of the TGF-β/Smad pathway (drawn by Figdraw).
Ds effectively attenuated nano-COM-induced HK-2 damage and delayed the EMT process by interfering with TGF-β expression and secretion and inhibiting the activation of the TGF-β/Smad pathway (drawn by Figdraw).