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Spectral evidence for Dirac spinons in a kagome lattice antiferromagnet

A Dirac quantum spin liquid phase is predicted to have a continuum of fractionalized spinon excitations with a Dirac cone dispersion. A spin continuum consistent with this picture has now been observed in neutron scattering measurements.

Zhenyuan Zeng
Chengkang Zhou
Shiliang Li
Research09 May 2024
Nature Physics

Volume: 20, P: 1097-1102
Dimensionality crossover to a two-dimensional vestigial nematic state from a three-dimensional antiferromagnet in a honeycomb van der Waals magnet

Magnetic phases that are stabilized by quantum fluctuations in low dimensions are rare. A thickness-dependent crossover from three-dimensional antiferromagnetism to a two-dimensional vestigial nematic state that is driven by fluctuations has now been observed.

Zeliang Sun
Gaihua Ye
Liuyan Zhao
Research26 Aug 2024
Nature Physics

Volume: 20, P: 1764-1771
Internal cost of spontaneous deception revealed by ERPs and EEG spectral perturbations

Chengkang Zhu
Jingjing Pan
Jianbiao Li
ResearchOpen Access01 Apr 2019
Scientific Reports

Volume: 9, P: 1-11
Lack of TRPV1 aggravates obesity-associated hypertension through the disturbance of mitochondrial Ca2+ homeostasis in brown adipose tissue

Li Li
Liqun Ma
Zhiming Zhu
ResearchOpen Access18 Jan 2022
Hypertension Research

Volume: 45, P: 789-801
TRPC5 deletion in the central amygdala antagonizes high-fat diet-induced obesity by increasing sympathetic innervation

Huan Ma
Chengkang He
Zhiming Zhu
Research20 May 2022
International Journal of Obesity

Volume: 46, P: 1544-1555

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Spectral evidence for Dirac spinons in a kagome lattice antiferromagnet

Dimensionality crossover to a two-dimensional vestigial nematic state from a three-dimensional antiferromagnet in a honeycomb van der Waals magnet

Internal cost of spontaneous deception revealed by ERPs and EEG spectral perturbations

Lack of TRPV1 aggravates obesity-associated hypertension through the disturbance of mitochondrial Ca2+ homeostasis in brown adipose tissue

TRPC5 deletion in the central amygdala antagonizes high-fat diet-induced obesity by increasing sympathetic innervation

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