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Showing 1–7 of 7 results
Advanced filters: Author: Richard G.A. Faragher Clear advanced filters

  • On 12 February 2025, a joint meeting of the UK Aging Networks was held in Liverpool, UK. It was convened by the ECMage (extracellular matrix aging) network and EuroAgeNet, an initiative led by ECMage but involving four other UK aging networks — namely, the building links in aging science and translation network (BLAST), the cognitive frailty interdisciplinary network (CFIN), the aging and nutrient sensing network (AGENTS) and the food systems for older people (Food4Years) network — together with industrial and European partners. In this Meeting Report, we summarize the opinions of an industrial panel and round-table discussions on barriers and opportunities related to academic–industrial partnerships.

    • Joe Swift
    • Angela Cucchi
    • Elizabeth G. Canty-Laird
    News & Views
    Nature Aging
    P: 1-5

  • Drugs that modulate fundamental mechanisms of ageing offer the promise of substantially improving the health of ageing populations, but innovative approaches to identify and evaluate such ‘gerotherapeutics’ are needed.

    • Richard G. A. Faragher
    • Richard C. Hartley
    Comments & Opinion
    Nature Reviews Drug Discovery
    Volume: 23, P: 725-726

  • The conservation of processes associated with normal ageing across species suggests that model systems can contribute to our understanding of human brain ageing. In this Perspective article the authors review the insights into longevity emerging from model organisms and highlight the need for the new paradigms in gerontology to be applied to the CNS.

    • Mark Yeoman
    • Greg Scutt
    • Richard Faragher
    Reviews
    Nature Reviews Neuroscience
    Volume: 13, P: 435-445

    • David Kipling
    • David Wynford-Thomas
    • Richard G.A. Faragher
    Correspondence
    Nature Biotechnology
    Volume: 17, P: 313

  • Telomeres have long been suspected to be involved in ageing, and we now have the first direct evidence. Transgenic mice were generated bearing a deletion in themTRgene, which is an essential component of telomerase, the enzyme that maintains telomeres. After six generations of inbreeding, newborn mice show many of the physiological changes associated with normal — but aged — mice.

    • David Kipling
    • Richard G. A. Faragher
    News & Views
    Nature
    Volume: 398, P: 191-193