Abstract
ENZYMES involved in the reduction of triphosphopyridine nucleotide (TPN) have been reported to be of primary importance in the resistance of erythrocytes to drug-induced hæmolytic conditions1. A hereditary defect in man associated with erythrocytes sensitive to drugs (primaquine) has been described in which there is a deficiency of hexose monophosphate pathway enzymes in the erythrocytes. Evidence of a susceptibility to drug-induced hæmolysis of erythrocytes has also been reported in the dog2.
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SMITH, J., BARNES, J., KANEKO, J. et al. Erythrocytic Enzymes of Various Animal Species. Nature 205, 298–299 (1965). https://doi.org/10.1038/205298a0
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DOI: https://doi.org/10.1038/205298a0
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