Fig. 4: Immune landscapes in DLBCL: inflamed phenotype, immune suppressed subtype.

Mutations in NFkB, NOTCH2, and NOTCH1 among others in DLBCL, contribute to excessive cytokine secretion, which in turn attracts T-cells, and enhances the inflammatory microenvironment. However, high numbers of suppressive immune cells inactivating class I HLA or CD58 in the tumor microenvironment, cause T-cell exhaustion.