Fig. 6

Proposed mechanistic depiction of sorafenib resistance. The alleviated ROS responses in sorafenib-resistant HCC cells was due to decreased mitochondrial biogenesis. Mechanistic dissection indicated that upregulation of UBQLN1 in sorafenib-resistant HCC cells accelerated the protein degradation of PGC1β, contributing to the decreased mitochondrial biogenesis and ROS generation, finally inducing sorafenib resistance