Fig. 6
From: Nucleic acid aptamers in orthopedic diseases: promising therapeutic agents for bone disorders

The mechanism of nucleic acid aptamers on chondrocytes in the pathological process of OA. a Schematic representation of the synthesis of tgg2-PEG2000-PAMAM6.0-Cy5.5. b Differentiated diffusion models of cationic nanocarriers binding to the negatively charged extracellular matrix (ECM) of chondrocytes via electrostatic interactions, followed by delivery into chondrocytes in normal and OA cartilage. c Modulation of the SIRT1/IL-6/STAT3 signaling pathway by miR-141/200c. Reproduced form ref. 410 with permission from Elsevier, copyright 2021