Fig. 8: Schematic depicting how G6PD status modulates the expressions of adhesion molecules, E-cadherin, and β-catenin, which play essential roles in regulating embryonic development

When the G6PD level is downregulated in cells, the cellular function of NOX is impaired, leading to the loss of redox signaling to induce the expressions of Smad3 and miR-200b. In such conditions, the expressions of E-cadherin and β-catenin are decreased, thus influencing the development of the embryo