Fig. 6: Schematic overview of the PI3K/Akt/BRCA1-A complex pathway in the Sei-1-transformed NIH-3T3 cell model

. Sei-1 activates the PI3K/Akt pathway independent of PTEN and inhibits the expression of the BRCA1-A complex to result in DSBs, leading to the formation of DMs in vivo. Sei-1 can also target the promoter and regulate the expression of BRCA1