Fig. 2: Prenatal GC exposure increased offspring’s myocardium infarct size and cardiomyocytes apoptosis after I/R in vivo. | Cell Death & Disease

Fig. 2: Prenatal GC exposure increased offspring’s myocardium infarct size and cardiomyocytes apoptosis after I/R in vivo.

From: The detrimental effects of glucocorticoids exposure during pregnancy on offspring’s cardiac functions mediated by hypermethylation of bone morphogenetic protein-4

Fig. 2

The TTC staining showed that myocardium infarct size was observably increased in prenatally DEX-exposed adult male offspring than that exposed to NS (a); TUNEL staining was performed on myocardium undergoing I/R. Although TUNEL-positive cells were rarely detected in NS group (b–d), DEX exposure led to much more extensive cardiomyocytes apoptosis (e-g). Nucleus stained with DAPI (blue)

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