Fig. 8: Schematic drawings for HSP60 and RPTOR protection against glucocorticoid deregulation of osteoblast autophagy and bone mass.

HSP60 is required to maintain osteoblast autophagy and osteogenesis. Glucocorticoid induces HSP60 loss escalating RPTOR de-phosphorylation, aggregation, and ubiquitination to deteriorate osteoblast autophagy and survival and ultimately speed up osteoporosis