Fig. 8: Schematic representation of the miR-185/Bgn/BMP-Smad1/5/8 signaling.

In this mechanism, during the osteogenic differentiation, the depletion of miR-185 upregulated Bgn expression, so as to activate the BMP/Smad1/5/8 signaling. The calvarial osteoblasts and mesenchymal stem cells (MSCs) of knockout (KO) mice exhibited increased osteogenic differentiation ability, and KO mice was protected during osteoporosis. miR-185, microRNA-185; Bgn, biglycan