Fig. 8: Graphical presentation of LL22NC03-N14H11.1/c-Myb/LTZR1/H-RAS/MAPK/DRP1 axis in HCC.

LL22NC03-N14H11.1 interacted with c-Myb to transcriptionally repress LZTR1, so as to decrease H-RAS (G12V) ubiquitination, activate MAPK pathway, and induce p-DRP1 (S616), resulting in induced mitochondrial fission and accelerated HCC progression.