Fig. 1: EEC–EMT is associated with ΔNp63α upregulation in endometrial biopsies from IUA patients.
From: ΔNp63α-induced DUSP4/GSK3β/SNAI1 pathway in epithelial cells drives endometrial fibrosis
a, b Representative images of ΔNp63α immunostaining in endometrial biopsies of normal (n = 30) and fibrotic endometria (n = 30). c, d Representative images of Masson staining in normal (n = 30) and fibrotic endometria (n = 30). e, f Representative images of E-cadherin immunostaining in endometrial biopsies of normal (n = 30) and fibrotic endometria (n = 30). g, h Representative images of N-cadherin immunostaining in endometrial biopsies of normal (n = 30) and fibrotic endometria (n = 30). i, j Representative images of Vimentin immunostaining in endometrial biopsies of normal (n = 30) and fibrotic endometria (n = 30). k, l Representative images of α-SMA immunostaining in endometrial biopsies of normal (n = 30) and fibrotic endometria (n = 30). Scale bars, 50 μm. Arrowhead: luminal epithelial cells; arrow: glandular epithelial cells. m Representative images of colocation of ΔNp63α with cytokeratin (CK), E-cadherin, N-cadherin, or α-SMA in endometrial biopsies of normal (n = 5) and fibrotic endometria (n = 5). Scale bars, 25 μm.
