Fig. 8: The diagram displaying the regulation of NFATc3 signaling by deSUMOylation under hypoxia.
From: Hypoxia-induced NFATc3 deSUMOylation enhances pancreatic carcinoma progression

Hypoxia resulted in SENP3-mediated deSUMOylation of NFATc3 at K384, suppressing the interaction between NFATc3 and its phosphokinase GSK-3β and subsequent increasing NFATc3 nuclear occupancy. In the nuclei of PDAC cells, NFATc3 formed complexes with other factors to coordinate target gene expression, which promoted PDAC progression.