Fig. 9: Summary schematic illustrating cognitive deficits induced by Seipin deficiency through impaired OPC differentiation, which is restored by RG administration.

Seipin deficiency impaired OPC differentiation, consequent myelination, and spatial cognition. Mechanistically, Seipin deficiency induced lipid dyshomeostasis via interfering expression of sphingolipid metabolism-related genes. RG administration rescued phenotypes induced by Seipin deficiency.