Correction to: Cell Death and Disease https://doi.org/10.1038/s41419-024-06748-w, published online 27 May 2024
In this article typos have been corrected:
-
1.
Consistently, the protective effects of FNDC5 on mitochondrial bioenergetics were counteracted in SIRT3 cKO mice.
-
2.
Figure legends: Fig. 1: a, Serum irisin levels were decreased in patients with poor outcomes compared to those with good outcomes (P < 0.001).
-
3.
The sentence ‘while there is no obvious change of acetylation level in DRP1’ has been deleted.
-
4.
Fig. 2: the MRI scan of the brain in the bottom panels of Fig. 2b were incorrect due to the mistaken images being inadvertently inserted during the assembly of Fig. 2 (separate attachments in e-mail named original Fig. 2 and amended Fig. 2).


The original article has been corrected.
Author information
Authors and Affiliations
Corresponding authors
Rights and permissions
Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.
About this article
Cite this article
Ge, Y., Wu, X., Cai, Y. et al. Correction: FNDC5 prevents oxidative stress and neuronal apoptosis after traumatic brain injury through SIRT3-dependent regulation of mitochondrial quality control. Cell Death Dis 15, 527 (2024). https://doi.org/10.1038/s41419-024-06901-5
Published:
Version of record:
DOI: https://doi.org/10.1038/s41419-024-06901-5