Fig. 4: METTL3-m6A-TGF-β signaling axis regulates cEndMT process in vitro. | Cell Death Discovery

Fig. 4: METTL3-m6A-TGF-β signaling axis regulates cEndMT process in vitro.

From: METTL3-m6A-mediated TGF-β signaling promotes Fuchs endothelial corneal dystrophy via regulating corneal endothelial-to-mesenchymal transition

Fig. 4: METTL3-m6A-TGF-β signaling axis regulates cEndMT process in vitro.

A Enrichment of differential m6A peaks in terms related to phenotypic transition shown by gene set enrichment analysis (GSEA). B Experimental design in vitro. C Western blot assay (left panel) and relative quantitative analysis of ZO-1 protein (middle panel) and α-SMA protein (right panel) in in vitro model with different treatments. D Immunofluorescence staining of ZO-1, Na+/K+-ATPase, α-SMA, and vimentin in in vitro model with different treatments. Data are presented as means ± SD from three independent experiments. *P < 0.05 versus si-NC groups.

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